NFKB1 and hepatitis A virus infection: Our results indicated salidroside pretreatment suppressed IκBα kinase and p65 phosphorylation in Con A-induced hepatitis, which in turn inhibited the activation of NF-κB. We speculated that the attenuation of the severity of Con A-induced hepatitis by salidroside pretreatment is mediated by suppression of inflammatory responses through downregulating phosphorylation of NF-κB activity partly.