KCNJ2 and Hypocholesterolemia: Finally, this study also raises the question as to whether (regardless of the distinct gain-of-function mutation causing SQT3S), hypocholesterolemia would contribute to trigger SQT3 arrhythmic episodes by further increasing Kir2.1 availability, or if, vice versa, borderline hypercholesterolemia would reduce the severity of symptoms.