CD8A and infection: We have previously shown in different models of mCMV infection, including HCT and adoptive CD8 T-cell transfer models as well as in the immunocompetent host, that control of acute pulmonary mCMV infection [11], [16], but also of infection at other sites, such as in liver parenchyma [17], [18], correlates with the formation of nodular inflammatory foci (NIF).