Interestingly, another group observed an upregulation of the miRNA processing machinery components (i.e., Dicer1, Drosha, and DGCR8), along with the expression of specific pri-miRNAs (such as pri-miR-7-1, pri-miR-16-2, and pri-miR-26a-1) in VAT samples from NASH, compared to non-NASH/NAFLD patients; hence, their findings suggest that VAT-derived miRNAs may contribute to the pathogenesis of NASH in obese subjects [40]. This evidence concerns the gene DICER1 and metabolic dysfunction-associated steatohepatitis.