On the other hand, several biological processes exhibited significantly high levels of perturbation upon infection with Mtb-H e.g. “stress-activated protein kinase signaling” pathway; NFkβ function e.g. “regulation of I-kappaB kinase/NF-kappaB cascade” and “positive regulation of I-kappaB kinase/NF-kappaB cascade”; processes involved in programmed cell death e.g. regulation of apoptosis. This evidence concerns the gene NFKB1 and infection.