Diabetic nephropathy and microalbuminurea have been found linked especially with postprandial hyperglycemia, among other glycemic parameters [102,103], whereas treatment with acarbose and other AGIs not only reduced postprandial hyperglycemia, but also microalbuminurea and related pro-inflammatory factors, such as interleukin 18 [103,104]. The gene discussed is IL18; the disease is Hyperglycemia.