ERG triggered the migratory potential of cancer cells by the overexpression of CXCR4 and ADAM metallopeptidase with a thrombospondin type 1 motif, 1 (ADAMTS1) as ChIP assay demonstrated direct binding of ERG to the promoter region for both CXCR4 and ADAMTS1[82]. Here, ADAMTS1 is linked to cancer.