Inflammatory markers such as interleukin-6 (IL-6), interleukin-1β (IL-1β), high sensitivity C-reactive protein (hs-CRP), and tumor necrosis factor-α (TNF-α) reflect the extent of myocardial necrosis and correlate with cardiac outcomes following MI [5–7]. The gene discussed is CRP; the disease is myocardial infarction.