Recently, the mechanism of action of FoxP3 in CD4+CD25+ T cells derived from BL/LL leprosy patients was shown to result from increased molecular interactions of FoxP3 with Histone deacetylases (HDAC7/9) in the nucleus of CD4+CD25+ T cells derived from BL/LL patients [24]. The gene discussed is FOXP3; the disease is leprosy.