Profibrotic stimuli such as Angiotensin II (AngII) or transforming growth factor β (TGF-β) treatment, hypertension and myocardial infarction lead to infiltration of inflammatory cells including macrophages, immune cells, neutrophils, mast cells and dendritic cells into the myocardium [7], [8], [9]. Here, AGT is linked to myocardial infarction.