Another postulated mechanism is that both tumours share the same aetiological factors, with mutation in the ataxia telangiectasia mutated (ATM) tumour suppressor gene[12], as well as infection with the Epstein-Barr virus (EBV)[13] and mouse mammary tumour virus (MMTV)[14] all currently implicated. The gene discussed is ATM; the disease is neoplasm.