Roeske et al. analyzed a discovery (N = 200) and replication (N = 186) cohort both selected for dyslexia and found association for a specific electrophysiological endophenotype of dyslexia (“mismatch negativity component” or MMN) (p = 5.14 × e−8 in combined dataset) pointing to the SLC2A3 gene, which is implicated in glucose transport in the brain [95]. The gene discussed is SLC2A3; the disease is dyslexia.