MYD88 and infection: This observation demonstrated a crucial crosstalk between miR-124 and MyD88 in modulate inflammatory response, and a feedback regulatory mechanism of miR-124/MyD88 in the alveolar epithelial cells against infection, suggesting that MyD88 is a critical mediator through which miR-124 exerts its biological functions in alveolar epithelial cells in response to mycobacterial infection.