In our study, we have found that the serum CXCL10 level in HFRS patients is substantially higher than its corresponding level in healthy donors (Figure 1(a)) and the content of CXCL10 in the acute phase of HFRS is 3 times higher than that in the convalescent phase (Figure 1(b)). This evidence concerns the gene CXCL10 and hemorrhagic fever with renal syndrome.