CXCL10 and hemorrhagic fever with renal syndrome: These results suggest that the increasing level of CXCL10 in the acute phase of HFRS may recruit CD14+ cells (monocytes) and the recruited CD14+ monocytes may produce excessive cytokines to be involved in the “cytokine storm.” Our presumption is consistent with the findings of Cheung et al. in their study on SARS-coronavirus [11].