CCR6 and pneumococcal meningitis: Using a mouse model of pneumococcal meningitis, we could demonstrate a significant function of CCL20 and CCR6 in pneumococcal meningitis: whenever the CCL20-CCR6 axis was disabled, either by blockage of CCL20 using an anti-CCL20 antibody or by using CCR6-deficient mice, we noted a significant reduction of the CSF pleocytosis.