More recently, IL-19 and IL-24 have been shown to have an unequivocal immunosuppressive role in the skin by inhibiting the production of IL-1β and IL-17 and promoting infection with Staphyloccus aureus[18], whereas IL-26 has been shown to be more pro-inflammatory, as it can induce cytokine production and Th17 cell generation in rheumatoid arthritis [19]. The gene discussed is IL19; the disease is infection.