TBXAS1 and non-small cell lung carcinoma: Coupled with the results that 24 h treatment of 18β-GA dose-dependently suppressed cell proliferation and induced apoptosis, the decrease of TxAS by 12 h treatment of 18β-GA suggests that the 18β-GA -induced inhibition of TxAS is an upstream event of growth inhibition and apoptosis in NSCLC, which is further supported by the following time course experiments.