Since we found that telmisartan could inhibit the proliferation of these cell lines, we examined the function of the antitumor effects of telmisartan through PPARγ in HHUA endometrial cancer cells in vitro, using a WST-1 assay with 2-day exposure to both telmisartan at 10 or 50 μM and a PPARγ antagonist, GW9662, at 10 μM. The gene discussed is PPARG; the disease is endometrial cancer.