Therefore, postprandial hypertriglyceridemia and visceral obesity may be responsible for the early onset of atherosclerosis, and it is speculated that changes in the production of adipocytokines such as adiponectin, TNF-α, resistin and PAI-1 would be responsible for the progression of atherosclerosis in PHT rabbits. This evidence concerns the gene TNF and hypertriglyceridemia.