The further confirmed molecular genetic screening of child clinically categorized as Dravet showed a heterozygous missense in exon 2 as p.S103G, which causes a serine to glycine substitution in N-terminus of SCN1A gene and results in Dravet syndrome (13). This evidence concerns the gene SCN1A and encephalopathy, progressive, early-onset, with brain edema and/or leukoencephalopathy.