Nevertheless, deregulation of redox metabolism in cancer cells caused by overexpression of oncogenes such as Ras [225], c-Myc [231], c-Abl [240], and Src [241] and growth factor receptors such as c-MET [230], insulin-like growth factor receptor [242], EGFR [243], and VEGF receptor [244] may render cancer cells more vulnerable than normal cells to oxidative stress-induced death [245,246]. This evidence concerns the gene MYC and cancer.