For cancer cells to sustain their rapid proliferation and gain a survival advantage, glycolysis has been demonstrated as a hot spot for infection-mediated metabolic reprogramming.19, 20, 21, 22, 23 We found that LMP1 could mediate the upregulation of several glycolytic genes, suggesting that oncogenic viruses might have evolved similar mechanisms to hijack critical cellular interaction networks and lead to the increased amounts of glucose delivery to proliferating cells to enhance their tumorigenic capacity. This evidence concerns the gene PDLIM7 and infection.