BCL2L1 and cancer: In A549 cancer lung cells, the H2O2 response induced by MJ increased the expression of the proapoptotic Bax and Bcl-Xs (although not being that of the antiapoptotic Bcl-2 and Bcl-xL proteins) and this led to apoptosis; this event was inhibited by catalase (a specific inhibitor of H2O2) [36].