Neutrophils that are primarily recruited by CXCL8 can exert antiviral immunity amongst others by the release of inflammatory cytokines (e.g., IL-12, TNFα) and chemokines (e.g., CCL2, CXCL8, CXCL9, CXCL10) that leads to further recruitment of immune cells to the site of infection [101]. The gene discussed is TNF; the disease is infection.