This delayed increase of IL-5 and IL-10 is matching previous observations made by others at the ‘late stage’ of infection, in human AE [37], [38], [39] and are in agreement with the data usually reported from the study of lymphocytes from experimentally infected mice [40]; this combined cytokine profile has been strongly linked to parasite evasion from the host immune response [27], [41]. This evidence concerns the gene IL10 and infection.