CCR2 and Guillain-Barre syndrome, familial: Since hematogenous monocytes/macrophages are predominantly CCR2+ [11] and are the most prevalent leukocyte subpopulation observed in peripheral nerves in AIDP and murine EAN models [9], [12], [13], we sought to deduce whether CCR2 is directly relevant to sm-EAN pathogenesis using gene knockout and pharmacologic blockade strategies.