TGFBR2 and nasopharyngeal carcinoma: Notably, miR-93 or siRNA-TGFβR2 could give rise to an increased TGF-β1 expression in CNE1 cells and an increased TGF-β1 secretion in the culture supernatants of CNE1 cells, whereas miR-93 inhibitor enabled inverse alterations in CNE2 cells (Additional file 2: Figure S9), suggesting a possible feedback loop from miR-93-mediated TGFβR2 to TGF-β1 existed in NPC cells.