TGFBR2 and nasopharyngeal carcinoma: Multiple studies have suggested the existence of direct [52,53] or indirect [54-57] crosstalk between TGF-β signaling and PI3K/Akt signaling, so we wonder whether PI3K/Akt pathway is also involved in NPC aggressiveness in the presence of miR-93-mediated TGFβR2 inactivation.