FGF23 and hypophosphatemia: Further studies are needed to elucidate the pathogenesis of persistent long-term post-transplant hypophosphatemia, as some studies have reported that persistent hyperparathyroidism has a greater influence than FGF-23, while others suggest that FGF-23 is the main driving force behind the persistent dysregulation of phosphate homeostasis (53, 55, 57).