FNTA and cystic fibrosis: AmrZ significantly repressed many genes in the pyochelin and pyoverdine synthesis operons, including ppyR. In addition, the Fe(III)-pyochelin receptor fptA and ferripyoverdine receptors fpvA and fpvB were all significantly repressed by AmrZ, (5.44, 2.01, and 1.45 fold, respectively), suggesting that reliance on the iron acquisition systems is reduced in strains where AmrZ is highly expressed, such as in mucoid isolates from the CF lung.