Since low grade of local and systemic inflammation is characteristic of all stages of atherosclerosis, multiple markers of inflammation have been intensively evaluated as potential risk factors for the development of coronary artery disease (CAD) and its complications, such as high-sensitivity C-reactive protein (hs-CRP), interleukin-6, fibrinogen, leukocyte and its subsets counts [1]–[6]. This evidence concerns the gene CRP and atherosclerosis.