The size of tumors produced and speed of their development did not depend solely on the presence of the PSCs, but also the expression levels of PSCs derived Galectin-1, because cancer cells that were implanted together with pancreatic cancer associated PSCs (hCaPSCs) that exhibited high levels of Galectin-1 resulted in larger volume and weight of tumors, than those that were implanted with PSCs from normal pancreas. This evidence concerns the gene LGALS1 and familial pancreatic carcinoma.