Genome-wide analyses have identified deregulated miRNA expression in human malignancies [149] and a potential dual role in tumor formation, highlighting that miRNAs can modulate oncogenic or tumor suppressor pathways, including p53, c-MYC, RAS, and BCR-ABL, while expression of miRNAs themselves can be regulated by oncogenes or tumor suppressors. Here, ABL1 is linked to neoplasm.