However, another group found that TLR2-deficient mice fed a choline-deficient amino acid (CDAA) diet had similar steatosis to wild-type mice, but decreased inflammatory cell infiltration and hepatocyte ballooning, suggesting that while steatosis is independent of TLR2 signaling, TLR2 appears to play a role in inflammation [44]. This evidence concerns the gene TLR2 and steatosis.