Although the underlying molecular mechanisms of breast cancer pathogenesis remain mainly undiscovered, multiple genetic and epigenetic alterations have been connected to breast cancer, including the activation of oncogenes (MYC, ERBB2 and CCND1) [3–5] and the mutation or deletion of tumor suppressor genes (TP53 and CDH1) [6,7]. The gene discussed is TP53; the disease is breast carcinoma.