Although the underlying molecular mechanisms of breast cancer pathogenesis remain mainly undiscovered, multiple genetic and epigenetic alterations have been connected to breast cancer, including the activation of oncogenes (MYC, ERBB2 and CCND1) [3–5] and the mutation or deletion of tumor suppressor genes (TP53 and CDH1) [6,7]. This evidence concerns the gene ERBB2 and breast carcinoma.