Despite the identification of groups with confirmed clinical HT/dipping relationships, suggesting our study population was representative of those groups, we have not managed to confirm that the genetic variations in the ACE genotype—insertion/deletion (rs17997552), rs1800764, rs4459609—and RGS2 rs2746071 genotype could be connected with the regulation of the BP and lead to the disturbances in both HT and/or non-dipping. This evidence concerns the gene RGS2 and hematocrit.