TNF and IgA glomerulonephritis: The pathogenic role of anti-TNFα treatment in the development of IgA glomerulonephritis in this case is suggested by (i) the improvement in clinical and laboratory abnormalities after drug withdrawal and initiation of corticosteroid therapy and (ii) the temporal relation of new onset glomerular disease to drug use with long standing psoriasis of many years and no prior renal disease.