In renal research, more evidence demonstrate that specific microRNAs alter renal physiology by changing expression patterns, mediating actions of TGF-β on renal fibrosis, affecting normal functions of MC, TEC, and podocyte, and inducing ECM deposition, podocyte dysfunction, and albuminuria during renal diseases [24, 28, 33, 34]. Here, TGFB1 is linked to renal fibrosis.