Moreover, IR often occurs along with IR of hepatic and muscle cells, increases of glycogen production and output, reduction of insulin-stimulated glucose uptake and its manifestations, postprandial hyperglycemia, and reductions of dystrophin-glycoprotein generation and storage, which results in a weakening of the suppressive effect of insulin on lipolysis (2,4,5). The gene discussed is INS; the disease is Hyperglycemia.