More widespread expression of hERG protein in tumors (e.g. colorectal or endometrial cancer) and cancerous cells (e.g. acute myologenous or chronic lymphocytic leukemia) [12] suggests potential significance of hERG-based anticancer therapy beyond glioblastoma that warrants further exploration, carefully considering the significance of the individual cellular context for regulation of cell death and proliferation. Here, KCNH2 is linked to glioblastoma.