AKT1 and cervical carcinoma: miR-214 upregulates expression of Bax, caspase-9, caspase-8, and caspase-3, enhances apoptosis and inhibits cell growth, and increases sensitivity to cisplatin by silencing Bcl2l2 expression [56], and miR-218 induces apoptosis, suppresses tumor growth, and increases sensitivity to cisplatin through the AKT-mTOR signaling pathway in cervical cancer cells [57].