Taken together, we speculate that the distinct “Th specific” cytokine profiles we observe reflect cytokine secretion by the transferred cells and the “background secretion” of IL-13 and IL-17 is provided by endogenous cells; a hypothesis where future studies in our model will be able to make contributions towards an increased understanding of the contribution of different Th subsets in antigen-specific skin inflammation. The gene discussed is IL17A; the disease is dermatitis.