Analysis of athymic (nude) mice injected subcutaneously with A431 cells, which strongly express both EGFR and SGLT-1 [37], showed that the tumor growth rate in mice co-treated with both DXR and BLF501 was similar to that in mice treated with DXR alone (p = 0.1836) (Figure 6), indicating that oral administration of BLF501 does not interfere with DXR antitumor activity. The gene discussed is EGFR; the disease is neoplasm.