This was evaluated by comparing the expression of eight genes, which included the three endogenous control genes (PPIA, TBP and GAPDH), three transcription factors involved in the late B-cell lymphopoiesis (XBP1, IRF4 and PRDM1), and two genes related to oncogenesis of MM (MGST1 and WHSC1) in six amplified and non-amplified CCLs [see Additional file 4]. Here, PRDM1 is linked to Miyoshi myopathy.