Moreover, in vitro chemotaxis assays revealed that whereas T cells from naive mice were unable to migrate in response to the CXCR3-ligand CXCL10, T cells from P. berghei ANKA-infected mice showed a 3-fold increase in their CXCL10-mediated chemotaxis (Hansen et al.2007), indicating that during malaria, T cells acquire the capacity to migrate in response to this chemokine. The gene discussed is CXCR3; the disease is malaria.