TRIP10 and cancer: This antitumoral function of ArgBP2 depends on its ability to control the multiple interactions of its numerous molecular partners, especially those involved in actin dynamics regulation such as WAVEs (wiskott-Aldrich syndrome protein containing Verprolin homology domain) [22], c-Abl (Abelson kinase) [23], CIP4 (Cdc42 interacting protein 4) [24], and PTP-PEST (Protein tyrosine phosphatase with PEST domain) [25], and has been observed in other type of cancers [26], [27], [28].