The enhanced expression of several lignin biosynthesis genes after infection with the tin2 mutant coincided with an transcriptional upregulation of several plant cell wall remodeling genes (Figure 12—source data 1) and a decrease in ferulic acid (Figure 4F), which is involved in crosslinking hemicellulosic polysaccharides and provides a nucleation site for lignification (Grabber et al., 2002), suggesting a connection between these processes. This evidence concerns the gene TINF2 and infection.