While studies here did not reveal a difference in the wound environment beyond small changes in leukocyte populations after PVA sponge implantation in MerTK−/− mice, a recent report found that MerTK-deficiency during myocardial infarction (MI) resulted in accumulation of apoptotic cardiomyocytes and subsequent adverse outcome [24]. This evidence concerns the gene MERTK and myocardial infarction.