Nevertheless, more recent reports showed transactivation of the EGFR by FPR1 in glioblastoma cells and in monocytes, in which FPR1 modulates the activation of EGFR and TrkA, the NGF receptor [32]–[33]; thus, we cannot exclude that the uPAR-mediated activation of EGFR observed in previous studies [26] involved also FPR1. The gene discussed is EGFR; the disease is glioblastoma.