SAA1 and endothelial dysfunction: Besides some evidence of an antiatherogenic role in promoting cholesterol efflux from cells [26–30], a plethora of studies have suggested proatherogenic effects for SAA, such as promotion of monocytes and neutrophils chemotaxis [31–33], macrophage foam cells formation [34], stimulation of proinflammatory cytokines secretion by monocytes-macrophages and lymphocytes [33, 35, 36], and induction of endothelial dysfunction [33, 37, 38].