Le Bourhis et al. (7) reported that MR1 molecules were induced to be overexpressed in fibroblasts and bone marrow-derived dendritic cells prior to bacterial infection as opposed to the natural overexpression of MR1 after infection observed in primary B cells and B-LCL used in our studies, as well as considerably higher MOI’s (up to 1,000:1) as compared with our study (0.3–200). Here, MR1 is linked to bacterial infectious disease.